A new scientific study has refuted earlier reports that daily cannabis use decreases the size of the brain. With long term alcohol use, a reduction in brain size is a side effect, I guess someone put two and two together and concluded cannabis must be as bad as alcohol. Alcohol is not a natural substance, marijuana is. Our bodies are first introduced to cannabinoids in our mothers breast milk. As the psychological layers of cynical government propaganda are peeled away, cannabis is being scientifically proven to be healthy for our bodies and minds. The previous studies, either through negligence or design, coupled alcohol use with cannabis use when testing for a change in the size of the brain. So if you don’t smoke tobacco or drink alcohol, then your brain will be perfectly healthy even if you like to consume some cannabis. In previous studies, THC has been shown to protect the brain, especially from serious accident brain traumas. Science report after science report points to marijuana in your system being the safer, healthier state to be in. How do you like them apples.
For the science geeks (yes I am one of them!) I am including the science extract from the journal of neuroscience below.
Recent research has suggested that marijuana use is associated with volumetric and shape differences in subcortical structures, including the nucleus accumbens and amygdala, in a dose-dependent fashion. Replication of such results in well controlled studies is essential to clarify the effects of marijuana. To that end, this retrospective study examined brain morphology in a sample of adult daily marijuana users (n = 29) versus nonusers (n = 29) and a sample of adolescent daily users (n = 50) versus nonusers (n = 50). Groups were matched on a critical confounding variable, alcohol use, to a far greater degree than in previously published studies. We acquired high-resolution MRI scans, and investigated group differences in gray matter using voxel-based morphometry, surface-based morphometry, and shape analysis in structures suggested to be associated with marijuana use, as follows: the nucleus accumbens, amygdala, hippocampus, and cerebellum. No statistically significant differences were found between daily users and nonusers on volume or shape in the regions of interest. Effect sizes suggest that the failure to find differences was not due to a lack of statistical power, but rather was due to the lack of even a modest effect. In sum, the results indicate that, when carefully controlling for alcohol use, gender, age, and other variables, there is no association between marijuana use and standard volumetric or shape measurements of subcortical structures.